IL-17 is increased in asthmatic airways and induces human bronchial fibroblasts to produce cytokines

2001945 citationsJournal Articlehybrid Open Access

Authors

Sophie Molet · Cegep de Sainte Foy

Qutayba Hamid · McGill University

Francis Davoineb · Cegep de Sainte Foy

Esra Nutku · McGill University

Rame Tahaa · Cegep de Sainte Foy

Nathalie Pagé · Cegep de Sainte Foy

Ron Olivenstein · Cegep de Sainte Foy

Abstract

Our results, which demonstrate for the first time that eosinophils are a potential source of IL-17 within asthmatic airways, suggest that IL-17 might have the potential to amplify inflammatory responses through the release of proinflammatory mediators such as alpha-chemokines.

Topics & Keywords

Asthma and respiratory diseases Pediatric health and respiratory diseases IL-33, ST2, and ILC Pathways

Publication Details

Published in: Journal of Allergy and Clinical Immunology

Volume 108, Issue 3, pp. 430-438

DOI: 10.1067/mai.2001.117929

Field-Weighted Citation Impact: 7.82

Command Palette

IL-17 is increased in asthmatic airways and induces human bronchial fibroblasts to produce cytokines

Authors

Abstract

Topics & Keywords

Publication Details