Exploratory Study of <i><scp>MYD</scp>88</i> L265P, Rare <i><scp>NLRP</scp>3</i> Variants, and Clonal Hematopoiesis Prevalence in Patients With Schnitzler Syndrome

A shared molecular mechanism accounting for the pathogenesis of inflammation in SchS remains elusive. Clonal hematopoiesis is not associated with other somatic mutations found in individuals with SchS or aCAPS.