<scp>IL</scp> ‐33 Drives Inflammatory Changes and Extracellular Trap Formation in Eosinophils Involving Oxidised <scp>LDL</scp> and Complement Pathways

20260 citationsJournal Articlehybrid Open Access

Authors

Emiko Matsuyama · Keio University

Jun Miyata · Keio University

Takao Mochimaru · Keio University

Ryuta Onozato · Keio University

Yo Otsu · Keio University

Hisashi Sasaki · National Defense Medical College

Keeya Sunata · Keio University

Shinichi Okuzumi ·

Abstract

IL-33, in conjunction with oxLDL and the complement cascade, induces inflammatory changes in eosinophils, promoting an ETosis-prone phenotype. These pathways represent potential therapeutic targets in refractory eosinophilic diseases.

Topics & Keywords

IL-33, ST2, and ILC Pathways Eosinophilic Esophagitis Asthma and respiratory diseases

Publication Details

Published in: Allergy

DOI: 10.1111/all.70313

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<scp>IL</scp> ‐33 Drives Inflammatory Changes and Extracellular Trap Formation in Eosinophils Involving Oxidised <scp>LDL</scp> and Complement Pathways

Authors

Abstract

Topics & Keywords

Publication Details