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Michael G. Schlossmacher | Researchclopedia

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Michael G. Schlossmacher

University of Ottawa ORCID

182

Works

15,865

Citations

56

h-index

82

i10-index

Research Topics

Parkinson's Disease Mechanisms and Treatments Nuclear Receptors and Signaling Neurological disorders and treatments

Affiliations

University of Kassel2006–2013

Boston University2006–2006

Brigham and Women's Hospital1990–2013

Harvard University1990–2013

Austrian Academy of Sciences1992–1992

Top Works

Amyloid β-peptide is produced by cultured cells during normal metabolism

Christian Haass,Michael G. Schlossmacher,et al.

19921,899 citations

Neuroinflammation and Neurodegeneration Mechanisms

Alzheimer's disease research and treatments

Phosphorylation of Ser-129 Is the Dominant Pathological Modification of α-Synuclein in Familial and Sporadic Lewy Body Disease

John P. Anderson,Donald E. Walker,et al.

20061,418 citations

Ubiquitination of a New Form of α-Synuclein by Parkin from Human Brain: Implications for Parkinson's Disease

Hideki Shimura,Michael G. Schlossmacher,et al.

20011,052 citations

Dopamine covalently modifies and functionally inactivates parkin

Matthew J. LaVoie,Beth L. Ostaszewski,et al.

2005714 citations

Detection of oligomeric forms of α‐synuclein protein in human plasma as a potential biomarker for Parkinson's disease

Omar M. A. El‐Agnaf,Sultan A. Salem,et al.

2006707 citations

α-Synuclein and tau concentrations in cerebrospinal fluid of patients presenting with parkinsonism: a cohort study

Brit Mollenhauer,Joseph J. Locascio,et al.

2011625 citations

Molecular markers of early Parkinson's disease based on gene expression in blood

Clemens R. Scherzer,Aron C. Eklund,et al.

2007513 citations

beta-Amyloid peptide and a 3-kDa fragment are derived by distinct cellular mechanisms.

Christian Haass,Albert Y. Hung,et al.

1993462 citations

Decreased α-synuclein in cerebrospinal fluid of aged individuals and subjects with Parkinson’s disease

Takahiko Tokuda,Sultan A. Salem,et al.

2006431 citations