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Mouadh Benamar | Researchclopedia

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Mouadh Benamar

Broad Institute

33

Works

575

Citations

10

h-index

10

i10-index

Research Topics

Advanced Breast Cancer Therapies Microtubule and mitosis dynamics Ubiquitin and proteasome pathways

Affiliations

Broad Institute2025–2025

Harvard University2025–2025

Precision for Medicine (United States)2024–2024

Dana-Farber Cancer Institute2023–2025

University of Virginia2014–2024

Top Works

Combined CDK4/6 and mTOR Inhibition Is Synergistic against Glioblastoma via Multiple Mechanisms

Inan Olmez,Breanna Brenneman,et al.

2017104 citations

DNA Repair Mechanisms

Genomics and Chromatin Dynamics

MicroDNA levels are dependent on MMEJ, repressed by c-NHEJ pathway, and stimulated by DNA damage

Teressa Paulsen,Pumoli Malapati,et al.

202195 citations

Inactivation of the CRL4-CDT2-SET8/p21 ubiquitylation and degradation axis underlies the therapeutic efficacy of pevonedistat in melanoma

Mouadh Benamar,Fadila Guessous,et al.

201675 citations

The Neddylation Inhibitor Pevonedistat (MLN4924) Suppresses and Radiosensitizes Head and Neck Squamous Carcinoma Cells and Tumors

Vanessa Vanderdys,Amir Allak,et al.

201773 citations

Combined c-Met/Trk Inhibition Overcomes Resistance to CDK4/6 Inhibitors in Glioblastoma

Inan Olmez,Ying Zhang,et al.

201864 citations

Chemotherapy-Induced Distal Enhancers Drive Transcriptional Programs to Maintain the Chemoresistant State in Ovarian Cancer

Stephen Shang,Jiekun Yang,et al.

201962 citations

Targeting Nucleophosmin 1 Represents a Rational Strategy for Radiation Sensitization

Konjeti R. Sekhar,Mouadh Benamar,et al.

201434 citations

Genomic mediators of acquired resistance to immunotherapy in metastatic melanoma

Julia Schiantarelli,Mouadh Benamar,et al.

202525 citations

The Barrier Molecules Junction Plakoglobin, Filaggrin, and Dystonin Play Roles in Melanoma Growth and Angiogenesis

Katie M. Leick,Anthony B. Rodriguez,et al.

201924 citations