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Albert W. Cheng | Researchclopedia

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Albert W. Cheng

Jackson Laboratory ORCID

98

Works

18,591

Citations

38

h-index

42

i10-index

Research Topics

CRISPR and Genetic Engineering Epigenetics and DNA Methylation Pluripotent Stem Cells Research

Affiliations

Salk Institute for Biological Studies2015–2015

University of Connecticut2016–2019

Chinese Academy of Sciences2024–2025

Hong Kong University of Science and Technology2005–2005

University of Rochester Medical Center2014–2014

Top Works

Histone H3K27ac separates active from poised enhancers and predicts developmental state

Menno P. Creyghton,Albert W. Cheng,et al.

20104,453 citations

Genomics and Chromatin Dynamics

RNA and protein synthesis mechanisms

One-Step Generation of Mice Carrying Mutations in Multiple Genes by CRISPR/Cas-Mediated Genome Engineering

Haoyi Wang,Hui Yang,et al.

20133,536 citations

One-Step Generation of Mice Carrying Reporter and Conditional Alleles by CRISPR/Cas-Mediated Genome Engineering

Hui Yang,Haoyi Wang,et al.

20131,612 citations

Human embryonic stem cells with biological and epigenetic characteristics similar to those of mouse ESCs

Jacob H. Hanna,Albert W. Cheng,et al.

2010845 citations

Single-Cell Expression Analyses during Cellular Reprogramming Reveal an Early Stochastic and a Late Hierarchic Phase

Yosef Buganim,Dina A. Faddah,et al.

2012844 citations

Multiplexed activation of endogenous genes by CRISPR-on, an RNA-guided transcriptional activator system

Albert W. Cheng,Haoyi Wang,et al.

2013760 citations

Tet1 Is Dispensable for Maintaining Pluripotency and Its Loss Is Compatible with Embryonic and Postnatal Development

Meelad M. Dawlaty,Kibibi Ganz,et al.

2011508 citations

Tet1 Is Critical for Neuronal Activity-Regulated Gene Expression and Memory Extinction

Andrii Rudenko,Meelad M. Dawlaty,et al.

2013450 citations

Combined Deficiency of Tet1 and Tet2 Causes Epigenetic Abnormalities but Is Compatible with Postnatal Development

Meelad M. Dawlaty,Achim Breiling,et al.

2013432 citations