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Kallyope (United States) | Researchclopedia

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Kallyope (United States)

New York, New Yorkcompany

36

Works

1,593

Citations

13

h-index

Top Research Topics

Diabetes Treatment and Management Pancreatic function and diabetes Receptor Mechanisms and Signaling

Top Researchers

Nancy A. Thornberry Kallyope (United States)

151 works40,096 citationsh-index: 73

Recent Research

Properties and functions of transcriptionally distinct enteric neurons

David E. Shi,Pranav Reddy,et al.

20252 citations

Regulation of Appetite and Obesity

Neuropeptides and Animal Physiology

Cancer Immunotherapy and Biomarkers

Speech Recognition and Synthesis

Genetic Associations and Epidemiology

Catalytic C–H Functionalization Methods

Gastrointestinal motility and disorders

Chromosomal and Genetic Variations

Catalytic Cross-Coupling Reactions

Robert P. J. Barretto Kallyope (United States)

17 works18,158 citationsh-index: 11

Yunhong Zhang Qilu University of Technology

665 works13,554 citationsh-index: 58

Andrea Peier Merck & Co., Inc., Rahway, NJ, USA (United States)

54 works8,833 citationsh-index: 26

Ji Zhang Shanghai University

233 works7,249 citationsh-index: 35

Xiao-Dong Yang Ningbo University

390 works6,731 citationsh-index: 47

Combined agonism of nutrient receptors <scp>GPR40</scp> and <scp>GPR119</scp> with K‐757 and K‐833 results in weight loss and blood pressure reductions in obese subjects without <scp>type 2 diabetes mellitus</scp>

Michael F. Crutchlow,Jiajun Liu,et al.

20251 citations

Combined <scp>GPR40</scp> and <scp>GPR119</scp> full agonism with K‐757 and K‐833 results in robust glucose lowering and modest weight loss in overweight/obese subjects with <scp>T2DM</scp>

Michael F. Crutchlow,Jiajun Liu,et al.

20251 citations

Discovery of Gut-Targeted GPR40 Agonist K-757 and GPR119 Agonist K-833, a Combination Treatment for Metabolic Disorders

Christopher R. Moyes,Shuwen He,et al.

20253 citations

Editorial: Obesity and type 2 diabetes mellitus: novel and alternative functional bioactive nutritional interventions

Renato Chaves Souto Branco,Luiz F. Barella,et al.

20240 citations

The gastrointestinal tract is a major source of the acute metformin-stimulated rise in GDF15

John W. R. Kincaid,Debra Rimmington,et al.

202414 citations